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Although the role of adipocyte-derived MTs is not fully understood, it is speculated that these stress-response and metal-binding proteins may act as antioxidants, shielding fatty acids from oxidative harm in adipocytes and during their transit from the micro-vasculature [18]
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This receptor interaction triggers intracellular signalling cascades, primarily the PI3K-AKT and MAPK pathways (1)
By blocking NNMT, 5-Amino-1MQ restores NAD+ availability in fat cells, shifting their behavior toward fat mobilization and oxidation rather than storage
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